Tissue Factor Modulates the Thrombogenicity of Human Atherosclerotic Plaques
- 4 February 1997
- journal article
- research article
- Published by Wolters Kluwer Health in Circulation
- Vol. 95 (3) , 594-599
- https://doi.org/10.1161/01.cir.95.3.594
Abstract
Background The thrombogenicity of a disrupted atherosclerotic lesion is dependent on the nature and extent of the plaque components exposed to flowing blood together with local rheology and a varie...Keywords
This publication has 23 references indexed in Scilit:
- Angiographic progression of coronary artery disease and the development of myocardial infarctionPublished by Elsevier ,2004
- Characterization of the relative thrombogenicity of atherosclerotic plaque components: Implications for consequences of plaque ruptureJournal of the American College of Cardiology, 1994
- Increased platelet deposition on atherosclerotic coronary arteries.Journal of Clinical Investigation, 1994
- Risk of thrombosis in human atherosclerotic plaques: role of extracellular lipid, macrophage, and smooth muscle cell content.Heart, 1993
- Angioscopic Evaluation of Coronary-Artery Thrombi in Acute Coronary SyndromesNew England Journal of Medicine, 1992
- Morphologic comparison of frequency and types of acute lesions in the major epicardial coronary arteries in unstable angina pectoris, sudden coronary death and acute myocardial infarctionJournal of the American College of Cardiology, 1991
- The role of platelets, thrombin and hyperplasia in restenosis after coronary angioplastyJournal of the American College of Cardiology, 1991
- Thrombogenicity of vascular cells. Comparison between endothelial cells isolated from different sources and smooth muscle cells and fibroblasts.Arteriosclerosis: An Official Journal of the American Heart Association, Inc., 1990
- Influence of arterial damage and wall shear rate on platelet deposition. Ex vivo study in a swine model.Arteriosclerosis: An Official Journal of the American Heart Association, Inc., 1986
- Plaque fissuring--the cause of acute myocardial infarction, sudden ischaemic death, and crescendo angina.Heart, 1985