Increased glucose transport by human fibroblasts with a heritable defect in insulin binding
- 1 July 1989
- journal article
- research article
- Published by Elsevier in Metabolism
- Vol. 38 (7) , 690-697
- https://doi.org/10.1016/0026-0495(89)90109-1
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Two Mutant Alleles of the Insulin Receptor Gene in a Patient with Extreme Insulin ResistanceScience, 1988
- Defects in Human Insulin Receptor Gene ExpressionMolecular Endocrinology, 1988
- After Insulin BindsScience, 1987
- Replacement of insulin receptor tyrosine residues 1162 and 1163 compromises insulin-stimulated kinase activity and uptake of 2-deoxyglucoseCell, 1986
- The human insulin receptor cDNA: The structural basis for hormone-activated transmembrane signallingCell, 1985
- Human insulin receptor and its relationship to the tyrosine kinase family of oncogenesNature, 1985
- Receptor defects in patients with extreme insulin resistanceDiabetes/Metabolism Research and Reviews, 1985
- Decreased Insulin Binding in Cultured Lymphocytes from Two Patients with Extreme Insulin Resistance*Journal of Clinical Endocrinology & Metabolism, 1982
- Cell Culture Studies on Patients with Extreme Insulin Resistance. II. Abnormal Biological Responses in Cultured Fibroblasts*Journal of Clinical Endocrinology & Metabolism, 1982
- Cell Culture Studies on Patients with Extreme Insulin Resistance. I. Receptor Defects on Cultured Fibroblasts*Journal of Clinical Endocrinology & Metabolism, 1982