Disruption of Smad4 in neural crest cells leads to mid-gestation death with pharyngeal arch, craniofacial and cardiac defects
- 15 April 2008
- journal article
- Published by Elsevier in Developmental Biology
- Vol. 316 (2) , 417-430
- https://doi.org/10.1016/j.ydbio.2008.02.006
Abstract
No abstract availableKeywords
This publication has 63 references indexed in Scilit:
- Smad4 is required to regulate the fate of cranial neural crest cellsDevelopmental Biology, 2007
- Smad signaling in the neural crest regulates cardiac outflow tract remodeling through cell autonomous and non-cell autonomous effectsDevelopmental Biology, 2007
- Mesodermal expression ofTbx1is necessary and sufficient for pharyngeal arch and cardiac outflow tract developmentDevelopment, 2006
- Fgf8 expression in the Tbx1 domain causes skeletal abnormalities and modifies the aortic arch but not the outflow tract phenotype of Tbx1 mutantsDevelopmental Biology, 2006
- Hematopoiesis Controlled by Distinct TIF1γ and Smad4 Branches of the TGFβ PathwayCell, 2006
- Cardiovascular malformations with normal smooth muscle differentiation in neural crest-specific type II TGFβ receptor (Tgfbr2) mutant miceDevelopmental Biology, 2006
- Patterning the neural crest derivatives during development of the vertebrate head: insights from avian studiesJournal of Anatomy, 2005
- New insights into TGF-β–Smad signallingPublished by Elsevier ,2004
- DiGeorge syndrome: an updateCurrent Opinion in Cardiology, 2004
- The contribution of both forebrain and midbrain crest cells to the mesenchyme in the frontonasal mass of mouse embryosDevelopmental Biology, 1994