Interactions ofChlamydia pneumoniaewith Human Endothelial Cells
Open Access
- 1 June 2000
- journal article
- review article
- Published by Oxford University Press (OUP) in The Journal of Infectious Diseases
- Vol. 181 (s3) , S479-S482
- https://doi.org/10.1086/315620
Abstract
In order to fulfill the “biological plausibility” criterion of a role for infection with Chlamydia pneumoniae in the pathogenesis of human atheroscleKeywords
This publication has 22 references indexed in Scilit:
- Chlamydia pneumoniae Infection of Vascular Smooth Muscle and Endothelial Cells Activates NF-κB and Induces Tissue Factor and PAI-1 ExpressionCirculation, 1999
- Endothelial Cytotoxicity Mediated by Serum Antibodies to Heat Shock Proteins of Escherichia coli and Chlamydia pneumoniaeCirculation, 1999
- Chlamydial and human heat shock protein 60s activate human vascular endothelium, smooth muscle cells, and macrophagesJournal of Clinical Investigation, 1999
- Chlamydia pneumoniae and Cardiovascular DiseaseEmerging Infectious Diseases, 1998
- Chlamydial Heat Shock Protein 60 Localizes in Human Atheroma and Regulates Macrophage Tumor Necrosis Factor-α and Matrix Metalloproteinase ExpressionCirculation, 1998
- Isolation ofChlamydia pneumoniaefrom a Carotid Endarterectomy SpecimenThe Journal of Infectious Diseases, 1997
- Isolation of Chlamydia pneumoniae from the Coronary Artery of a Patient with Coronary AtherosclerosisAnnals of Internal Medicine, 1996
- Expression of adhesion molecules on endothelial cells stimulated byChlamydia pneumoniaeMicrobial Pathogenesis, 1996
- Chlamydia pneumoniae multiplies in human endothelial cells in vitroMicrobial Pathogenesis, 1994
- The pathogenesis of atherosclerosis: a perspective for the 1990sNature, 1993