Misrejoining of DNA double-strand breaks in primary and transformed human and rodent cells: a comparison between the HPRT region and other genomic locations
- 1 April 1999
- journal article
- research article
- Published by Elsevier in Mutation Research/DNA Repair
- Vol. 433 (3) , 193-205
- https://doi.org/10.1016/s0921-8777(99)00008-7
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Homology-directed repair is a major double-strand break repair pathway in mammalian cellsProceedings of the National Academy of Sciences, 1998
- Unequal homologous recombination between LINE-1 elements as a mutational mechanism in human genetic disease 1 1Edited by J. KarnJournal of Molecular Biology, 1998
- Interstitial deletions and intrachromosomal amplification initiated from a double-strand break targeted to a mammalian chromosomeThe EMBO Journal, 1998
- CONSEQUENCES OF CHROMOSOMAL ABNORMALITIES IN TUMOR DEVELOPMENTAnnual Review of Genetics, 1997
- Chromosomal Double-Strand Breaks Induce Gene Conversion at High Frequency in Mammalian CellsMolecular and Cellular Biology, 1997
- Highly conservative reciprocal translocations formed by apparent joining of exchanged DNA double-strand break endsProceedings of the National Academy of Sciences, 1997
- A breakpoint map of recurrent chromosomal rearrangements in human neoplasiaNature Genetics, 1997
- Chromosomal translocations in human cancerNature, 1994
- Illegitimate recombination induced by DNA double-strand breaks in a mammalian chromosome.Molecular and Cellular Biology, 1994
- Formation of large deletions by illegitimate recombination in the HPRT gene of primary human fibroblasts.Proceedings of the National Academy of Sciences, 1993