Interferon-γ Inducible Protein (IP-10) Expression Is Mediated by CD8+ T Cells and Is Regulated by CD4+ T Cells During the Elicitation of Contact Hypersensitivity
- 1 September 1996
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 107 (3) , 360-366
- https://doi.org/10.1111/1523-1747.ep12363337
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- T cell populations primed by hapten sensitization in contact sensitivity are distinguished by polarized patterns of cytokine production: interferon gamma-producing (Tc1) effector CD8+ T cells and interleukin (Il) 4/Il-10-producing (Th2) negative regulatory CD4+ T cells.The Journal of Experimental Medicine, 1996
- Regulation of Chemokine Gene Expression by Contact Hypersensitivity and by Oral ToleranceaAnnals of the New York Academy of Sciences, 1996
- Production of the RANTES chemokine in delayed-type hypersensitivity reactions: involvement of macrophages and endothelial cells.The Journal of Experimental Medicine, 1994
- L3T4 (CD4+) cells that mediate contact sensitivity to trinitrochlorobenzene express I-A determinantsCellular Immunology, 1991
- Properties of the Novel Proinflammatory Supergene "Intercrine" Cytokine FamilyAnnual Review of Immunology, 1991
- Keratinocytes as initiators of inflammationThe Lancet, 1991
- A macrophage LPS-inducible early gene encodes the murine homologue of IP-10Biochemical and Biophysical Research Communications, 1990
- Neutrophil-activating peptide-1/interleukin 8, a novel cytokine that activates neutrophils.Journal of Clinical Investigation, 1989
- Biochemical characterization of a gamma interferon-inducible cytokine (IP-10).The Journal of Experimental Medicine, 1987
- Therapy with monoclonal antibodies by elimination of T-cell subsets in vivoNature, 1984