A Common Mechanism Mediates Long-Term Changes in Synaptic Transmission after Chronic Cocaine and Morphine
- 1 March 1996
- Vol. 16 (3) , 631-639
- https://doi.org/10.1016/s0896-6273(00)80082-3
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Cocaine inhibits GABA release in the VTA through endogenous 5-HTJournal of Neuroscience, 1994
- Potentiation of cAMP responses by metabotropic glutamate receptors depresses excitatory synaptic transmission by a kinase-independent mechanismNeuron, 1994
- Dopamine D1 receptors facilitate transmitter releaseNature, 1993
- Ecto‐5′‐Nucleotidase Is Associated with Cholinergic Nerve Terminals in the Hippocampus but Not in the Cerebral Cortex of the RatJournal of Neurochemistry, 1992
- Decreased activity of rat A10 dopamine neurons following withdrawal from repeated cocaineEuropean Journal of Pharmacology, 1992
- Primary sequence of cyclic nucleotide phosphodiesterase isozymes and the design of selective inhibitorsTrends in Pharmacological Sciences, 1990
- Use of microdialysis to measure brain noradrenergic receptor function in vivoBrain Research, 1988
- The Physiological Role of Adenosine In The Central Nervous SystemPublished by Elsevier ,1985
- Dendritic release of dopamine in the substantia nigraNature, 1981
- Release of dopamine from dendrites in rat substantia nigraNature, 1976