Exertional Rhabdomyolysis

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Abstract
Acute disintegration of muscle and myoglobinuria after intense, prolonged or repetitive physical exertion has become a widely recognized clinical entity. It appears that such rhabdomyolysis may occur in normal men provided they are under sufficient physical stress. Indeed, highly specialized laboratory technics suggest that detectable quantities of myoglobin appear consistently in serum and urine after extreme muscular activity or trauma such as that incurred during participation in contact sports.1 Moreover, in untrained but not in trained men, intense exercise is consistently followed by a rise of CPK, SGOT, and LDH activity in serum to abnormally high values, presumably reflecting skeletal . . .

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