The Effects of Norepinephrine and Angiotensine on the Lymphatic System

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Abstract
The maximal end pressure within the artificially obstructed thoracic duct can be enhanced by injecting norepinephrine or Hypertensin. The tone of the isolated thoracic duct, as well as that of the isolated external jugular vein can be increased by means of norepinephrine and Hypertensin. The decreased lymph flow in the thoracic duct, accompanying the increased blood pressure evoked by norepinephrine and Hypertensin, is not due to a decreased lymph production; on the contrary, the production of the lymph in the abdominal cavity and in the lower extremities is increased during this period. Decreased lymph flow is a result of the enhanced tone of the thoracic duct. Phleboconstriction caused by norepinephrine and by Hypertensin is responsible both for the increased lymph production and, after cessation of the arterial vasoconstriction, for the slowing down of the passage of the lymph.

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