Bcl-xL Inhibits Cytochrome c Release but Not Mitochondrial Depolarization during the Activation of Multiple Death Pathways by Tumor Necrosis Factor-α
Open Access
- 1 October 2000
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 275 (40) , 31546-31553
- https://doi.org/10.1074/jbc.m001363200
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Death Receptors: Signaling and ModulationScience, 1998
- Mitochondria and ApoptosisScience, 1998
- Two CD95 (APO-1/Fas) signaling pathwaysThe EMBO Journal, 1998
- Induction of Apoptotic Program in Cell-Free Extracts: Requirement for dATP and Cytochrome cPublished by Elsevier ,1996
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell DeathCell, 1996
- Mitochondrial control of nuclear apoptosis.The Journal of Experimental Medicine, 1996
- Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death.The Journal of Experimental Medicine, 1995
- FADD, a novel death domain-containing protein, interacts with the death domain of fas and initiates apoptosisCell, 1995
- Cell-free apoptosis in Xenopus egg extracts: Inhibition by Bcl-2 and requirement for an organelle fraction enriched in mitochondriaCell, 1994