Is pathogenic humoral autoimmunity a Th1 response? Lessons from (for) myasthenia gravis
- 1 January 2000
- journal article
- review article
- Published by Elsevier in Immunology Today
- Vol. 21 (1) , 19-23
- https://doi.org/10.1016/s0167-5699(99)01553-4
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Interferon γ (IFN-γ) Is Necessary for the Genesis of Acetylcholine Receptor–induced Clinical Experimental Autoimmune Myasthenia gravis in MiceThe Journal of Experimental Medicine, 1997
- Interleukin-4 Protects against a Genetically Linked Lupus-like Autoimmune SyndromeThe Journal of Experimental Medicine, 1997
- Autoimmune T cells in myasthenia gravis: heterogeneity and potential for specific immunotargetingImmunology Today, 1996
- Studies on the interdependence of gp39 and B7 expression and function during antigen‐specific immune responsesEuropean Journal of Immunology, 1995
- Th1 and Th2 CD4+ T cells in the pathogenesis of organ-specific autoimmune diseasesImmunology Today, 1995
- CD27: marker and mediator of T-cell activation?Immunology Today, 1994
- The Nicotinic Acetylcholine Receptor: Structure and Autoimmune PathologyCritical Reviews in Biochemistry and Molecular Biology, 1994
- T Cell Reactivity to Acetylcholine Receptor in Rats Orally Tolerized against Experimental Autoimmune Myasthenia GravisCellular Immunology, 1993
- Immunosuppression in Vivo by a Soluble Form of the CTLA-4 T Cell Activation MoleculeScience, 1992
- Experimental autoimmune myasthenia: A model of myasthenia gravis in rats and guinea pigs.The Journal of Experimental Medicine, 1975