Interactions at a dioxin responsive element (DRE) and an overlapping κB site within the hs4 domain of the 3′α immunoglobulin heavy chain enhancer

This publication has 45 references indexed in Scilit:

• 2,3,7,8-Tetrachlorodibenzo-p-dioxin, an Exogenous Modulator of the 3′α Immunoglobulin Heavy Chain Enhancer in the CH12.LX Mouse Cell Line
  The Journal of Pharmacology and Experimental Therapeutics, 2004
• Ah receptor and NF-κB interactions: mechanisms and physiological implications
  Chemico-Biological Interactions, 2002
• Aryl Hydrocarbon Receptor-Deficient Mice Generate Normal Immune Responses to Model Antigens and Are Resistant to TCDD-Induced Immune Suppression
  Toxicology and Applied Pharmacology, 2001
• The RelA NF-κB subunit and the aryl hydrocarbon receptor (AhR) cooperate to transactivate the c-myc promoter in mammary cells
  Oncogene, 2000
• NF-κB Activity Is Required for the Deregulation of c-myc Expression by the Immunoglobulin Heavy Chain Enhancer
  Published by Elsevier ,2000
• Distinct Domains of the RelA NF-κB Subunit Are Required for Negative Cross-talk and Direct Interaction with the Glucocorticoid Receptor
  Published by Elsevier ,1997
• THE NF-κB AND IκB PROTEINS: New Discoveries and Insights
  Annual Review of Immunology, 1996
• Identification of a locus control region in the immunoglobulin heavy-chain locus that deregulates c-myc expression in plasmacytoma and Burkitt's lymphoma cells.
  Genes & Development, 1994
• Function and Activation of NF-kappaB in the Immune System
  Annual Review of Immunology, 1994
• Spontaneous murine B-cell leukaemia
  Nature, 1978