Increase in extracellular serotonin produced by uptake inhibitors is enhanced after chronic treatment with fluoxetine
- 1 April 1994
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 171 (1-2) , 183-186
- https://doi.org/10.1016/0304-3940(94)90635-1
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- Comparative effects of chronic 8-OH-DPAT, gepirone and ipsapirone treatment on the sensitivity of somatodendritic 5-HT1A autoreceptorsNeuropharmacology, 1993
- Serotonin 5‐HT1A Autoreceptor Blockade Potentiates the Ability of the 5‐HT Reuptake Inhibitor Citalopram to Increase Nerve Terminal Output of 5‐HT In Vivo: A Microdialysis StudyJournal of Neurochemistry, 1993
- Alteration of 5‐HT1A receptor binding sites following chronic treatment with ipsapirone measured by quantitative autoradiographySynapse, 1992
- Citalopram's ability to increase the extracellular concentrations of serotonin in the dorsal raphe prevents the drug's effect in the frontal cortexBrain Research, 1992
- In vivo calibration of microdialysis probes for exogenous compoundsAnalytical Chemistry, 1992
- Effect of fluoxetine on serotonin and dopamine concentration in microdialysis fluid from rat striatumLife Sciences, 1992
- Fluoxetine, a selective inhibitor of serotonin uptakeMedicinal Research Reviews, 1991
- Modifications of the Serotonin System by Antidepressant TreatmentsJournal of Clinical Psychopharmacology, 1987
- 8-hydroxy-2-(di-n-propylamino)tetralin, 8-OH-DPAT, a potent and selective simplified ergot congener with central 5-HT-receptor stimulating activityJournal Of Neural Transmission-Parkinsons Disease and Dementia Section, 1982
- Raphe neurons: effect of tricyclic antidepressant drugsBrain Research, 1972