Differential effects of kindled and electrically induced seizures on a glutamate receptor (GluR1) gene expression
- 1 March 1993
- journal article
- Published by Elsevier in Epilepsy Research
- Vol. 14 (3) , 221-227
- https://doi.org/10.1016/0920-1211(93)90046-a
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Limbic seizure and brain damage produced by kainic acid: Mechanisms and relevance to human temporal lobe epilepsyPublished by Elsevier ,2003
- Cloning of a putative glutamate receptor: A low affinity kainate-binding subunitNeuron, 1992
- Cloning of a cDNA for a glutamate receptor subunit activated by kainate but not AMPANature, 1991
- Molecular Cloning and Functional Expression of Glutamate Receptor Subunit GenesScience, 1990
- Limbic Seizures Increase Neuronal Production of Messenger RNA for Nerve Growth FactorScience, 1989
- Retardation of amygdala kindling by antagonism of NMD-aspartate and muscarinic cholinergic receptors: Evidence for the summation of excitatory mechanisms in kindlingExperimental Neurology, 1988
- Ketamine-induced changes in kindled amygdaloid seizuresNeuropharmacology, 1983
- Phencyclidine inhibition of the rate of development of amygdaloid kindled seizuresExperimental Neurology, 1982
- Pharmacological modification of amygdaloid-kindled seizuresNeuropharmacology, 1980
- A permanent change in brain function resulting from daily electrical stimulationExperimental Neurology, 1969