Vitamin D3-up-regulated Protein-1 Is a Stress-responsive Gene That Regulates Cardiomyocyte Viability through Interaction with Thioredoxin
Open Access
- 1 July 2002
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 277 (29) , 26496-26500
- https://doi.org/10.1074/jbc.m202133200
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Reactive Oxygen Species in Mechanical Stress-Induced Cardiac HypertrophyBiochemical and Biophysical Research Communications, 2001
- Series Introduction: The transcription factor NF-κB and human diseaseJournal of Clinical Investigation, 2001
- A Possible Interaction of Thioredoxin with VDUP1 in HeLa Cells Detected in a Yeast Two-Hybrid SystemBiochemical and Biophysical Research Communications, 2000
- Distinct Roles of Thioredoxin in the Cytoplasm and in the NucleusJournal of Biological Chemistry, 1999
- Oxidative stress activates extracellular signal-regulated kinases through Src and Ras in cultured cardiac myocytes of neonatal rats.Journal of Clinical Investigation, 1997
- Protection Against Reperfusion-Induced Arrhythmias by Human ThioredoxinJournal of Cardiovascular Pharmacology, 1996
- Endothelin-1 Is Involved in Mechanical Stress-induced Cardiomyocyte HypertrophyJournal of Biological Chemistry, 1996
- Mismatch Binding Protein-Based Mutation Detection SystemsMethods, 1995
- Autocrine release of angiotensin II mediates stretch-induced hypertrophy of cardiac myocytes in vitroCell, 1993
- Oxygen-derived free radicals and postischemic myocardial dysfunction (“stunned myocardium”)Journal of the American College of Cardiology, 1988