Vitamin C blocks vascular dysfunction and release of interleukin-6 induced by endothelin-1 in humans in vivo
- 28 February 2007
- journal article
- Published by Elsevier in Atherosclerosis
- Vol. 190 (2) , 408-415
- https://doi.org/10.1016/j.atherosclerosis.2006.02.018
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Activation of Pro-Inflammatory and Anti-Inflammatory Cytokines in Host Organs During Chronic Allograft Rejection: Role of Endothelin Receptor SignalingAmerican Journal of Transplantation, 2005
- Endothelin receptor blockade improves endothelial function in atherosclerotic patients on angiotensin converting enzyme inhibitionJournal of Internal Medicine, 2005
- Endothelium-Restricted Overexpression of Human Endothelin-1 Causes Vascular Remodeling and Endothelial DysfunctionCirculation, 2004
- Interleukin-6 Induces Oxidative Stress and Endothelial Dysfunction by Overexpression of the Angiotensin II Type 1 ReceptorCirculation Research, 2004
- Endothelin-1 Stimulates Arterial VCAM-1 Expression Via NADPH Oxidase-Derived Superoxide in Mineralocorticoid HypertensionHypertension, 2003
- NADPH Oxidase–Derived Superoxide Augments Endothelin-1–Induced Venoconstriction in Mineralocorticoid HypertensionHypertension, 2003
- Endothelin-1 Increases Vascular Superoxide via Endothelin A –NADPH Oxidase Pathway in Low-Renin HypertensionCirculation, 2003
- Inflammation in atherosclerosisNature, 2002
- Endothelin-1 Induces NAD(P)H Oxidase in Human Endothelial CellsBiochemical and Biophysical Research Communications, 2000
- Interleukin-6 as a central mediator of cardiovascular risk associated with chronic inflammation, smoking, diabetes, and visceral obesity: down-regulation with essential fatty acids, ethanol and pentoxifyllineMedical Hypotheses, 1999