Chronic myelomonocytic leukemia: Tel-a-kinase what Ets all about
- 22 April 1994
- Vol. 77 (2) , 171-173
- https://doi.org/10.1016/0092-8674(94)90307-7
Abstract
No abstract availableThis publication has 16 references indexed in Scilit:
- The nuclear tyrosine kinase c-abl negatively regulates cell growthCell, 1994
- Fusion of PDGF receptor β to a novel ets-like gene, tel, in chronic myelomonocytic leukemia with t(5;12) chromosomal translocationCell, 1994
- Fusion of a Kinase Gene, ALK , to a Nucleolar Protein Gene, NPM , in Non-Hodgkin's LymphomaScience, 1994
- A coiled-coil oligomerization domain of Bcr is essential for the transforming function of Bcr-Abl oncoproteins.Molecular and Cellular Biology, 1993
- Dimerization mediated through a leucine zipper activates the oncogenic potential of the met receptor tyrosine kinase.Molecular and Cellular Biology, 1993
- Platelet-derived growth factor receptor mediates activation of ras through different signaling pathways in different cell types.Molecular and Cellular Biology, 1993
- Molecular characterization of a thyroid tumor-specific transforming sequence formed by the fusion of ret tyrosine kinase and the regulatory subunit RI alpha of cyclic AMP-dependent protein kinase A.Molecular and Cellular Biology, 1993
- Mutations of the ras protooncogenes in chronic myelogenous leukemia: a high frequency of ras mutations in bcr/abl rearrangement-negative chronic myelogenous leukemiaBlood, 1989
- Interactions between heterologous helix-loop-helix proteins generate complexes that bind specifically to a common DNA sequenceCell, 1989
- Mechanism of activation of the human trk oncogene.Molecular and Cellular Biology, 1989