Chk1 complements the G2/M checkpoint defect and radiosensitivity of ataxia-telangiectasia cells
- 7 January 1999
- journal article
- Published by Springer Nature in Oncogene
- Vol. 18 (1) , 249-256
- https://doi.org/10.1038/sj.onc.1202257
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 49 references indexed in Scilit:
- THE GENETIC DEFECT IN ATAXIA-TELANGIECTASIAAnnual Review of Immunology, 1997
- 14-3-3 ζ Protein Binds to the Carboxyl Half of Mouse Wee1 KinaseBiochemical and Biophysical Research Communications, 1997
- Defect in Multiple Cell Cycle Checkpoints in Ataxia-Telangiectasia PostirradiationPublished by Elsevier ,1996
- Relationship of the ataxia-telangiectasia protein ATM to phosphoinositide 3-kinaseTrends in Biochemical Sciences, 1995
- p53 is required for radiation-induced apoptosis in mouse thymocytesNature, 1993
- Feedback control of mitosis in budding yeastCell, 1991
- Effect of Caffeine on γ-ray-induced G2 Delay in Ataxia TelangiectasiaInternational Journal of Radiation Biology, 1985
- Comparison of kinetics of X-ray-induced cell killing in normal, ataxia telangiectasia and hereditary retinoblastoma fibroblastsMutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 1983
- Cell Death, Chromosome Damage and Mitotic Delay in Normal Human, Ataxia Telangiectasia and Retinoblastoma Fibroblasts after X-irradiationInternational Journal of Radiation Biology and Related Studies in Physics, Chemistry and Medicine, 1981
- DNA synthesis and cell cycle progression of synchronized L-cells after irradiation in various phases of the cell cycleRadiation and Environmental Biophysics, 1980