Role of Tumor Necrosis Factor-α in Neuronal and Glial Apoptosis after Spinal Cord Injury
- 1 November 2000
- journal article
- research article
- Published by Elsevier in Experimental Neurology
- Vol. 166 (1) , 190-195
- https://doi.org/10.1006/exnr.2000.7494
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Early neuronal expression of tumor necrosis factor-α after experimental brain injury contributes to neurological impairmentJournal of Neuroimmunology, 1999
- Oligodendrocyte Apoptosis and Primary Demyelination Induced by Local TNF/p55TNF Receptor Signaling in the Central Nervous System of Transgenic MiceThe American Journal of Pathology, 1998
- Review : Apoptosis and Spinal Cord InjuryThe Neuroscientist, 1998
- Neuronal and Glial Apoptosis after Traumatic Spinal Cord InjuryJournal of Neuroscience, 1997
- ICE/CED‐3 Family Executes Oligodendrocyte Apoptosis by Tumor Necrosis FactorJournal of Neurochemistry, 1997
- Apoptosis and delayed degeneration after spinal cord injury in rats and monkeysNature Medicine, 1997
- An endogenous 55 kDa TNF receptor mediates cell death in a neural cell lineMolecular Brain Research, 1996
- TNFα gene expression is induced in neurones after a hippocampal lesionNeuroReport, 1994
- The Microglial Reaction in the Rat Dorsal Hippocampus following Transient Forebrain IschemiaJournal of Cerebral Blood Flow & Metabolism, 1991
- Production of tumor necrosis factor and other cytokines by astrocytes stimulated with lipopolysaccharide or a neurotropic virus.Proceedings of the National Academy of Sciences, 1989