Overexpression of mitochondrial 3-hydroxy-3-methylglutaryl-CoA synthase in transgenic mice causes hepatic hyperketogenesis.
Open Access
- 1 March 1994
- journal article
- abstracts
- Published by Elsevier
- Vol. 269 (9) , 6267-6270
- https://doi.org/10.1016/s0021-9258(17)37364-7
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Control of hepatic mitochondrial 3‐hydroxy‐3‐methylglutaryl‐CoA synthase during the foetal/neonatal transition, suckling and weaning in the ratEuropean Journal of Biochemistry, 1991
- Glucagon activates mitochondrial 3‐hydroxy‐3‐methylglutaryl‐CoA synthase in vivo by decreasing the extent of succinylation of the enzymeEuropean Journal of Biochemistry, 1990
- Transgenic Mice with I-A on Islet Cells Are Normoglycemic But Immunologically IntolerantScience, 1989
- Intramitochondrial factors controlling hepatic fatty acid oxidation at weaning in the ratFEBS Letters, 1988
- Mechanisms of regulation of the partition of fatty acids between oxidation and esterification in the liverProgress in Lipid Research, 1984
- Inhibition of transcription of the phosphoenolpyruvate carboxykinase gene by insulinNature, 1983
- Regulation of Hepatic Fatty Acid Oxidation and Ketone Body ProductionAnnual Review of Biochemistry, 1980
- Isolation of biologically active ribonucleic acid from sources enriched in ribonucleaseBiochemistry, 1979
- Ultrastructural and biochemical studies of isolated adult rat hepatocytes prepared under hypoxic conditions Cryopreservation of hepatocytesExperimental Cell Research, 1976
- Fuels, Hormones, and Liver Metabolism at Term and during the Early Postnatal Period in the RatJournal of Clinical Investigation, 1973