28S ribosome degradation in lymphoid cell apoptosis: evidence for caspase and Bcl-2-dependent and -independent pathways
- 28 September 2000
- journal article
- Published by Springer Nature in Cell Death & Differentiation
- Vol. 7 (10) , 994-1001
- https://doi.org/10.1038/sj.cdd.4400731
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- The ability to cleave 28S ribosomal RNA during apoptosis is a cell-type dependent trait unrelated to DNA fragmentationCell Death & Differentiation, 1997
- Accumulation of Sequence-specific RNA-binding Proteins in the Cytosol of Activated T Cells Undergoing RNA Degradation and ApoptosisPublished by Elsevier ,1995
- Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosisNature, 1995
- Selective cleavage of 28S rRNA variable regions V3 and V13 in myeloid leukemia cell apoptosisFEBS Letters, 1993
- γ-Ray-induced Transcription and Apoptosis-associated Loss of 28S rRNA in Interphase Human LymphocytesInternational Journal of Radiation Biology, 1993
- Apoptosis: Mechanisms and Roles in PathologyPublished by Elsevier ,1991
- Glucocorticoid regulation of two serine hydrolases in rat splenic lymphocytes in vitroBiochimica et Biophysica Acta (BBA) - General Subjects, 1981
- Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activationNature, 1980
- Independence of θ and TL surface antigens and killing by thymidine, cortisol, phytohemagglutinin, and cyclic AMP in a murine lymphomaBiochemical and Biophysical Research Communications, 1973
- Differentiated functions expressed by cultured mouse lymphoma cellsExperimental Cell Research, 1970