Abstract
Pharmacologic modulation of the immediate and late phase reaction (LPR) to anti-human IgE was further investigated in a double-blind cross-over study. Tranexamine acid (AMCA) 1 g t.i.d. [2 times/day] 24 h prior to and following intradermal injection of anti-IgE produced .apprx. 40% inhibition of the LPR (P < 0.01) without affecting the early response as compared with placebo in 10 volunteers. Antagonistic effect on activation of fibrinolysis and possibly the complement system is suggested as a possible mode of action.