Expression of p16INK4aSuppresses the Unbounded and Anchorage-Independent Growth of a Glioblastoma Cell Line That Lacks p16INK4a
- 24 February 1997
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 231 (3) , 743-750
- https://doi.org/10.1006/bbrc.1997.6181
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Compilation of somatic mutations of theCDKN2 gene in human cancers: Non-random distribution of base substitutionsGenes, Chromosomes and Cancer, 1996
- Differences in Substrate Specificity between Cdk2-Cyclin A and Cdk2-Cyclin E in VitroBiochemical and Biophysical Research Communications, 1995
- Inhibitors of mammalian G1 cyclin-dependent kinases.Genes & Development, 1995
- D-type cyclinsTrends in Biochemical Sciences, 1995
- The retinoblastoma protein and cell cycle controlCell, 1995
- Cell-cycle regulators and cancerTrends in Genetics, 1995
- pl5INK4B is a potentia| effector of TGF-β-induced cell cycle arrestNature, 1994
- A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4Nature, 1993
- Direct binding of cyclin D to the retinoblastoma gene product (pRb) and pRb phosphorylation by the cyclin D-dependent kinase CDK4Genes & Development, 1993
- The retinoblastoma protein as a transcriptional repressorTrends in Cell Biology, 1993