Myocardial malondialdehyde and uric acid release after short-lasting coronary occlusions during coronary angioplasty: Potential mechanisms for free radical generation
- 1 August 1991
- journal article
- Published by Elsevier in The American Journal of Cardiology
- Vol. 68 (4) , 392-395
- https://doi.org/10.1016/0002-9149(91)90838-c
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Oxygen-derived free radicals and postischemic myocardial dysfunction (“stunned myocardium”)Journal of the American College of Cardiology, 1988
- Free radicals and myocardial injury: pharmacologic implications.Circulation, 1986
- Superoxide dismutase plus catalase improve contractile function in the canine model of the "stunned myocardium".Circulation Research, 1986
- Oxygen-Derived Free Radicals in Postischemic Tissue InjuryNew England Journal of Medicine, 1985
- Free-radical mechanisms in tissue injuryBiochemical Journal, 1984
- Amphipathic metabolites and membrane dysfunction in ischemic myocardium.Circulation Research, 1984
- Left ventricular performance, regional blood flow, wall motion, and lactate metabolism during transluminal angioplasty.Circulation, 1984
- Canine myocardial reperfusion injury. Its reduction by the combined administration of superoxide dismutase and catalase.Circulation Research, 1984
- Localization of xanthine oxidase in mammary-gland epithelium and capillary endotheliumCell, 1981
- Prolonged depletion of ATP and of the adenine nucleotide pool due to delayed resynthesis of adenine nucleotides following reversible myocardial ischemic injury in dogsJournal of Molecular and Cellular Cardiology, 1981