Hypoxia increases production of interleukin-1 and tumor necrosis factor by human mononuclear cells
- 31 May 1991
- Vol. 3 (3) , 189-194
- https://doi.org/10.1016/1043-4666(91)90015-6
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- In vitro production of IL 1β, IL 1α, TNF and IL 2 in healthy subjects: distribution, effect of cyclooxygenase inhibition and evidence of independent gene regulationEuropean Journal of Immunology, 1989
- C5a stimulates secretion of tumor necrosis factor from human mononuclear cells in vitro. Comparison with secretion of interleukin 1 beta and interleukin 1 alpha.The Journal of Experimental Medicine, 1988
- Regulation of human lung fibroblast glycosaminoglycan production by recombinant interferons, tumor necrosis factor, and lymphotoxin.Journal of Clinical Investigation, 1988
- Smooth Muscle-Mediated Connective Tissue Remodeling in Pulmonary HypertensionScience, 1987
- Cachectin: More Than a Tumor Necrosis FactorNew England Journal of Medicine, 1987
- Shock and Tissue Injury Induced by Recombinant Human CachectinScience, 1986
- Interleukin 1 acts on cultured human vascular endothelium to increase the adhesion of polymorphonuclear leukocytes, monocytes, and related leukocyte cell lines.Journal of Clinical Investigation, 1985
- Additive effect of intravascular complement activation and brief episodes of hypoxia in producing increased permeability in the rabbit lung.Journal of Clinical Investigation, 1985
- Isolation of biologically active ribonucleic acid from sources enriched in ribonucleaseBiochemistry, 1979
- Phorbol myristic acetate stimulates LAF production by the macrophage cell line, P388DCellular Immunology, 1978