SHP-1 deficiency and increased inflammatory gene expression in PBMCs of multiple sclerosis patients
Open Access
- 21 January 2008
- journal article
- research article
- Published by Elsevier in Laboratory Investigation
- Vol. 88 (3) , 243-255
- https://doi.org/10.1038/labinvest.3700720
Abstract
No abstract availableKeywords
This publication has 92 references indexed in Scilit:
- Negative regulation of the SH2-homology–containing protein-tyrosine phosphatase-1 (SHP-1) P2 promoter by the HTLV-1 Tax oncoproteinBlood, 2007
- B Cell-Specific Deletion of Protein-Tyrosine Phosphatase Shp1 Promotes B-1a Cell Development and Causes Systemic AutoimmunityImmunity, 2007
- PU.1 Activates Transcription of SHP-1 Gene in Hematopoietic CellsJournal of Biological Chemistry, 2007
- IL‐4 induces expression of TARC/CCL17 via two STAT6 binding sitesEuropean Journal of Immunology, 2006
- Regulation of avoidant behaviors and pain by the anti-inflammatory tyrosine phosphatase SHP-1Neuron Glia Biology, 2006
- Inverse regulation of inducible nitric oxide synthase (iNOS) and arginase I by the protein tyrosine phosphatase SHP-1 in CNS gliaGlia, 2006
- The expression and function of chemokines involved in CNS inflammationTrends in Pharmacological Sciences, 2006
- Reduced Expression and Promoter Methylation p16 Gene in Epstein‐Barr Virus‐associated Gastric CarcinomaJapanese Journal of Cancer Research, 2002
- Evolution of Autoantibody Responses via Somatic Hypermutation Outside of Germinal CentersScience, 2002
- The gene encoding hematopoietic cell phosphatase (SHP-1) is structurally and transcriptionally intact in polycythemia veraOncogene, 1997