Effects of estrogen, SU‐9055, and cyproterone acetate on the hypersecretory activity in the seminal vesicles of the castrate catfish, Heteropneustes fossilis (Bloch)

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Abstract
The effect of estradiol benzoate (EB), SU‐9055, and cyproterone acetate (CA) on the hypersecretory seminal vesicles of the castrate catfish has been studied. EB to a marked extent, SU‐9055 and CA to a lesser extent inhibit castration induced hypersecretion in the seminal vesicles. This implies that while EB inhibits the gonadotrophin production, SU‐9055 and CA act at the level of the interrenal glands and the seminal vesicles respectively. The data indicate that following castration the high level of circulating gonadotrophin induces the interrenals to produce androgens which in turn bring about hypersecretion in the seminal vesicles.

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