The fibrosis and atrial fibrillation: Is the transforming growth factor-β1 a candidate etiology of atrial fibrillation
- 31 December 2008
- journal article
- Published by Elsevier in Medical Hypotheses
- Vol. 70 (2) , 317-319
- https://doi.org/10.1016/j.mehy.2007.04.046
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Transforming Growth Factor-β, Cell Signaling and Cardiovascular DisordersCurrent Vascular Pharmacology, 2005
- Effect of Angiotensin II Receptor Blocker on Plasma Levels of TGF-β1 and Interstitial Fibrosis in Hypertensive Kidney Transplant PatientsAmerican Journal of Nephrology, 2003
- Alterations of β-adrenergic signaling and cardiac hypertrophy in transgenic mice overexpressing TGF-β1American Journal of Physiology-Heart and Circulatory Physiology, 2002
- Add-on angiotensin II receptor blockade lowers urinary transforming growth factor-β levelsAmerican Journal of Kidney Diseases, 2002
- Induction of Cardiac Fibrosis by Transforming Growth Factor-β1Molecular Genetics and Metabolism, 2000
- Angiotensin II directly increases transforming growth factor β1 and osteopontin and indirectly affects collagen mRNA expression in the human heartCardiovascular Research, 2000
- Atrial but Not Ventricular Fibrosis in Mice Expressing a Mutant Transforming Growth Factor-β 1 Transgene in the HeartCirculation Research, 2000
- Spontaneous Initiation of Atrial Fibrillation by Ectopic Beats Originating in the Pulmonary VeinsNew England Journal of Medicine, 1998
- Early Induction of Transforming Growth Factor-β via Angiotensin II Type 1 Receptors Contributes to Cardiac Fibrosis Induced by Long-term Blockade of Nitric Oxide Synthesis in RatsHypertension, 1998
- Evidence for reentry as a mechanism of cardiac arrhythmiasPublished by Springer Nature ,1975