Neurological impairment in fetal mouse brain by drinking water disinfectant byproducts
- 31 August 2005
- journal article
- Published by Elsevier in NeuroToxicology
- Vol. 26 (4) , 633-640
- https://doi.org/10.1016/j.neuro.2004.11.001
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Oxidative stress as a mediator of apoptosisPublished by Elsevier ,2002
- Drinking Water Contaminants and Adverse Pregnancy Outcomes: A ReviewEnvironmental Health Perspectives, 2002
- Induction of oxidative stress and TNF-α secretion by Dichloroacetonitrile, a water disinfectant by-product, as possible mediators of apoptosis or necrosis in a murine macrophage cell line (RAW)Toxicology in Vitro, 2000
- Chloroacetonitrile (CAN) induces glutathione depletion and 8-hydroxylation of guanine bases in rat gastric mucosaJournal of Biochemical and Molecular Toxicology, 1999
- Drinking water disinfection byproducts: review and approach to toxicity evaluation.Environmental Health Perspectives, 1999
- Programmed cell death is a universal feature of embryonic and postnatal neuroproliferative regions throughout the central nervous systemJournal of Comparative Neurology, 1998
- Effect of glutathione modulation on molecular interaction of [14C]-chloroacetonitrile with maternal and fetal DNA in miceReproductive Toxicology, 1993
- Studies on the mechanism of haloacetonitriles toxicity: quantitative whole body autoradiographic distribution of [2-14C]chloroacetonitrile in ratsToxicology, 1991
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976
- An improved cupric-silver method for impregnation of axonal and terminal degenerationBrain Research, 1971