Macrophage foam cells from human aortic fatty streaks take up β-VLDL and acetylated LDL in primary culture
- 1 October 1989
- journal article
- research article
- Published by Elsevier in Atherosclerosis
- Vol. 79 (2-3) , 173-182
- https://doi.org/10.1016/0021-9150(89)90122-6
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Malondialdehyde alteration of low density lipoproteins leads to cholesteryl ester accumulation in human monocyte-macrophagesProceedings of the National Academy of Sciences, 1980
- Cholesteryl ester accumulation in macrophages resulting from receptor-mediated uptake and degradation of hypercholesterolemic canine beta-very low density lipoproteins.Journal of Biological Chemistry, 1980
- Quantification of low density lipoprotein binding and cholesterol accumulation by single human fibroblasts using fluorescence microscopy.Journal of Lipid Research, 1980
- ARTERIAL FOAM CELLS WITH DISTINCTIVE IMMUNOMORPHOLOGIC AND HISTOCHEMICAL FEATURES OF MACROPHAGES1980
- Altered Metabolism (In Vivo and In Vitro) of Plasma Lipoproteins after Selective Chemical Modification of Lysine Residues of the ApoproteinsJournal of Clinical Investigation, 1979
- Characterization of low density lipoprotein-like particle in the human aorta from grossly normal and atherosclerotic regionsBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1979
- CHARACTERIZATION OF LIPID-LADEN AORTIC-CELLS FROM CHOLESTEROL-FED RABBITS .4. INVESTIGATION OF MACROPHAGE-LIKE PROPERTIES OF AORTIC CELL-POPULATIONS1979
- Binding site on macrophages that mediates uptake and degradation of acetylated low density lipoprotein, producing massive cholesterol depositionProceedings of the National Academy of Sciences, 1979
- CHARACTERIZATION OF LIPID-LADEN AORTIC CELLS FROM CHOLESTEROL-FED RABBITS .1. RESOLUTION OF AORTIC CELL-POPULATIONS BY METRIZAMIDE DENSITY GRADIENT CENTRIFUGATION1977
- Degradation of cationized low density lipoprotein and regulation of cholesterol metabolism in homozygous familial hypercholesterolemia fibroblasts.Proceedings of the National Academy of Sciences, 1976