Variations in ATM Protein Expression During Normal Lymphoid Differentiation and Among B-Cell-Derived Neoplasias
- 31 August 2003
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 163 (2) , 423-432
- https://doi.org/10.1016/s0002-9440(10)63672-3
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- ATM mutations are associated with inactivation of theARF-TP53 tumor suppressor pathway in diffuse large B-cell lymphomaBlood, 2002
- Sensing of intermediates in V(D)J recombination by ATMGenes & Development, 2002
- Epidermal Growth Factor Sensitizes Cells to Ionizing Radiation by Down-regulating Protein Mutated in Ataxia-TelangiectasiaJournal of Biological Chemistry, 2001
- Immature Thymocytes Undergoing Receptor Rearrangements Are Resistant to an Atm-Dependent Death Pathway Activated in Mature T Cells by Double-Stranded DNA BreaksThe Journal of Experimental Medicine, 2000
- Ionizing radiation activates the ATM kinase throughout the cell cycleOncogene, 2000
- DNA-PK, the DNA-activated protein kinase, is differentially expressed in normal and malignant human tissuesOncogene, 1999
- Upregulation of ATM in sclerosing adenosis of the breast [published erratum appears in Mol Pathol 1998 Dec;51(6):342]Molecular Pathology, 1998
- A gene transcribed from the bidirectional ATM promoter coding for a serine rich protein: amino acid sequence, structure and expression studiesHuman Molecular Genetics, 1996
- V(D)J recombination activates a p53-dependent DNA damage checkpoint in scid lymphocyte precursors.Genes & Development, 1996
- High Frequency and Error-prone DNA Recombination in Ataxia Telangiectasia Cell LinesPublished by Elsevier ,1996