Norrin, Frizzled-4, and Lrp5 Signaling in Endothelial Cells Controls a Genetic Program for Retinal Vascularization
Top Cited Papers
- 1 October 2009
- Vol. 139 (2) , 285-298
- https://doi.org/10.1016/j.cell.2009.07.047
Abstract
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This publication has 47 references indexed in Scilit:
- Wnt/β-catenin signaling is required for CNS, but not non-CNS, angiogenesisProceedings of the National Academy of Sciences, 2009
- Comparisons of structural and functional abnormalities in mouse b‐wave mutantsThe Journal of Physiology, 2008
- Vascular changes in the cerebellum of Norrin /Ndph knockout mice correlate with high expression of Norrin and Frizzled‐4European Journal of Neuroscience, 2008
- The Optokinetic Reflex as a Tool for Quantitative Analyses of Nervous System Function in Mice: Application to Genetic and Drug-Induced VariationPLOS ONE, 2008
- A model for familial exudative vitreoretinopathy caused by LPR5 mutationsHuman Molecular Genetics, 2008
- Sox17 is essential for the specification of cardiac mesoderm in embryonic stem cellsProceedings of the National Academy of Sciences, 2007
- Role of the Norrie Disease Pseudoglioma Gene in Sprouting Angiogenesis during Development of the Retinal VasculatureInvestigative Opthalmology & Visual Science, 2005
- Norrie disease and peripheral venous insufficiencyBritish Journal of Ophthalmology, 2004
- Tie2-Cre Transgenic Mice: A New Model for Endothelial Cell-Lineage Analysis in VivoDevelopmental Biology, 2001
- Modulation of Endothelial Cell Adhesion by Hevin, an Acidic Protein Associated with High Endothelial VenulesJournal of Biological Chemistry, 1996