Potassium Wasting and Other Renal Tubular Defects with Rifampin Nephrotoxicity

Abstract
Interstitial nephritis consequent to rifampin was associated with K wasting, an acidifying defect, high fractional uric acid excretion, and glucosuria, indicating a multiplicity of renal tubular transport abnormalities. Enlarged kidneys on sonogram and proteinuria were also observed.

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