A note on the utility of repair‐deficient st mus302 drosophila females in detecting chromosome loss and sex‐linked recessive lethals induced in the male genome by methyl methanesulfonate (MMS) and dimethylnitrosamine (DMN)
- 1 January 1982
- journal article
- Published by Wiley in Environmental Mutagenesis
- Vol. 4 (3) , 291-293
- https://doi.org/10.1002/em.2860040312
Abstract
No abstract availableThis publication has 7 references indexed in Scilit:
- Induced chromosom loss following treatment of postmeiotic cells of the Drosophila melanogaster male with MMS and DMN and matings with repair-proficient females and the repair-deficient females mei-9a and st mus302Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 1982
- Preliminary data suggesting that females of the repair-deficient strain designated st mus302 are strong potentiators of chromosome loss induced by procarbazine and diethylnitrosamine (DEN) in the male genome of DrosophilaMutation Research Letters, 1982
- Review of the current status of the mei-9a test for chromosome loss in Drosophila melanogaster: An assay with radically improved detection capacity for chromosome lesions induced by methyl methanesulfonate (MMS), dimenthylnitrosamine (DMN), and especially diethylnitrosamine (DEN) and procarbazineMutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 1981
- Mutants partially defective in excision repair at five autosomal loci in Drosophila melanogasterChromosoma, 1981
- CHARACTERIZATION OF POSTREPLICATION REPAIR IN MUTAGEN-SENSITIVE STRAINS OF DROSOPHILA MELANOGASTERGenetics, 1976
- THE mei-9a MUTANT OF DROSOPHILA MELANOGASTER INCREASES MUTAGEN SENSITIVITY AND DECREASES EXCISION REPAIRGenetics, 1976
- GENETIC ANALYSIS OF SEX CHROMOSOMAL MEIOTIC MUTANTS IN DROSOPHILA MELANOGASTERGenetics, 1972