H2A.Z Stabilizes Chromatin in a Way That Is Dependent on Core Histone Acetylation

This publication has 59 references indexed in Scilit:

• Histone Variant H2A.Z Marks the 5′ Ends of Both Active and Inactive Genes in Euchromatin
  Cell, 2005
• Genome-Wide Dynamics of Htz1, a Histone H2A Variant that Poises Repressed/Basal Promoters for Activation through Histone Loss
  Cell, 2005
• New twists on H2A.Z: a histone variant with a controversial structural and functional past
  Biochemistry and Cell Biology, 2004
• The Many Tales of a Tail: Carboxyl-Terminal Tail Heterogeneity Specializes Histone H2A Variants for Defined Chromatin Function
  Biochemistry, 2002
• Histone variant H2A.Z is required for early mammalian development
  Current Biology, 2001
• A Histone Variant, Htz1p, and a Sir1p-like Protein, Esc2p, Mediate Silencing at HMR
  Molecular Cell, 2000
• Regions of variant histone His2AvD required for Drosophila development
  Nature, 1999
• Essential and Nonessential Histone H2A Variants in Tetrahymena thermophila
  Molecular and Cellular Biology, 1996
• Characterization of the Proximal Promoter of the Human Histone H2A.Z Gene
  DNA and Cell Biology, 1995
• A histone variant, H2AvD, is essential in Drosophila melanogaster.
  Molecular Biology of the Cell, 1992