Smad and AML Proteins Synergistically Confer Transforming Growth Factor β1 Responsiveness to Human Germ-line IgA Genes

This publication has 48 references indexed in Scilit:

• Engineering hybrid genes without the use of restriction enzymes: gene splicing by overlap extension
  Published by Elsevier ,2003
• Downstream factors in transforming growth factor-β family signaling
  Molecular and Cellular Endocrinology, 1998
• Intracellular signaling of osteogenic protein-1 through Smad5 activation
  Journal of Cellular Physiology, 1998
• Human Smad3 and Smad4 Are Sequence-Specific Transcription Activators
  Published by Elsevier ,1998
• TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4
  The EMBO Journal, 1997
• The Missing Bone
  Cell, 1997
• The human I_α1 region contains a TGF–β1 responsive enhancer and a putative recombination hotspot
  International Immunology, 1995
• The human l_α1 and l_α2 germline promoter elements: proximal positive and distal negative elements may regulate the tissue specific expression of C_α1 and C_α2 germline transcripts
  International Immunology, 1993
• Interleukin 10 and transforming growth factor beta cooperate to induce anti-CD40-activated naive human B cells to secrete immunoglobulin A.
  The Journal of Experimental Medicine, 1992
• Transforming growth factor beta specifically enhances IgA production by lipopolysaccharide-stimulated murine B lymphocytes.
  The Journal of Experimental Medicine, 1989