Chronic cocaine administration sensitizes behavioral but not neuroendocrine responses
- 15 March 1991
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 543 (2) , 301-306
- https://doi.org/10.1016/0006-8993(91)90041-s
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Attenuation of amphetamine-stereotypy by mesostriatal dopamine depletion enhances plasma corticosterone: Implications for stereotypy as a coping responseBehavioral and Neural Biology, 1989
- Pure uptake blockers of dopamine can reduce prolactin secretion: Studies with diclofensineLife Sciences, 1988
- Cocaine stimulates adrenocorticotropin (ACTH) secretion through a corticotropin-releasing factor (CRF)-mediated mechanismBrain Research, 1987
- Cocaine induced secretion of ACTH, beta-endorphin, and corticosteronePeptides, 1987
- Enduring changes in brain and behavior produced by chronic amphetamine administration: A review and evaluation of animal models of amphetamine psychosisBrain Research Reviews, 1986
- Differences in Hypothalamo-Pituitary-Adrenocortical Activity in the Rat after Acute and Prolonged Treatment with MorphineNeuroendocrinology, 1984
- Repeated administration of pergolide to rats attenuates the acute elevation of serum corticosterone by pergolidePharmacology Biochemistry and Behavior, 1981
- Interchangeability of Stress and Amphetamine in SensitizationScience, 1980
- Lack of a high affinity transport system for dopamine in the median eminence and posterior pituitaryBrain Research, 1979
- Effects of amphetamine, methylphenidate and cocaine on serum prolactin concentrations in the male ratLife Sciences, 1977