Abstract
The respiratory metabolism was measured before, during, and after acute cardiac tamponade in 13 unanesthetized dogs. The O2 uptake was severely depressed during tamponade, and during recovery there was an excess O2 consumption. The O2 debt was calculated by totalling the O2 missed during tamponade and by estimating the O2 equivalent of the fixed acids produced in tamponade. Comparison of O2 lack with the excess O2 of recovery varied considerably from expt. to expt. The efficiency of recovery was much lower than that reported after muscular work. This suggests a possible difference in the removal of lactate in tamponade from that in severe exercise.

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