Female embryonic lethality in mice nullizygous for both Msh2 and p53
- 1 September 1997
- journal article
- research article
- Published by Springer Nature in Nature Genetics
- Vol. 17 (1) , 114-118
- https://doi.org/10.1038/ng0997-114
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Mutations in the p53 and SCID genes cooperate in tumorigenesis.Genes & Development, 1996
- MSH2 deficient mice are viable and susceptible to lymphoid tumoursNature Genetics, 1995
- Inactivation of the mouse Msh2 gene results in mismatch repair deficiency, methylation tolerance, hyperrecombination, and predisposition to cancerCell, 1995
- Deficiency of p53 accelerates mammary tumorigenesis in Wnt-1 transgenic mice and promotes chromosomal instability.Genes & Development, 1995
- X chromosome inactivation and the Xist geneCurrent Opinion in Genetics & Development, 1994
- Tumor spectrum analysis in p53-mutant miceCurrent Biology, 1994
- Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumoursNature, 1992
- A genetic model for colorectal tumorigenesisCell, 1990
- Differentiation of X chromosomes in early female mouse embryosExperimental Cell Research, 1974
- Asynchronous Duplication of Chromosomes in Cultured Cells of Chinese HamsterThe Journal of cell biology, 1960