Reduced dopamine output in the nucleus accumbens but not in the medial prefrontal cortex in rats displaying a mecamylamine-precipitated nicotine withdrawal syndrome
- 13 January 1998
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 779 (1-2) , 214-225
- https://doi.org/10.1016/s0006-8993(97)01135-9
Abstract
No abstract availableKeywords
This publication has 54 references indexed in Scilit:
- Mesolimbic Dopamine Activation—The Key to Nicotine Reinforcement?Published by Wiley ,2007
- Non-psychostimulant drugs of abuse and anxiogenic drugs activate with differential selectivity dopamine transmission in the nucleus accumbens and in the medial prefrontal cortex of the ratPsychopharmacology, 1996
- Increase of extracellular dopamine in the medial prefrontal cortex during spontaneous and naloxone-precipitated opiate abstinencePsychopharmacology, 1995
- Desensitization of the nicotine‐induced mesolimbic dopamine responses during constant infusion with nicotineBritish Journal of Pharmacology, 1995
- Decreased activity of rat A10 dopamine neurons following withdrawal from repeated cocaineEuropean Journal of Pharmacology, 1992
- Depression of Mesolimbic Dopamine Transmission and Sensitization to Morphine During Opiate AbstinenceJournal of Neurochemistry, 1992
- The influence of stress on psychopharmacological responses to nicotineBritish Journal of Addiction, 1991
- Differential Effect of Stress on In Vivo Dopamine Release in Striatum, Nucleus Accumbens, and Medial Frontal CortexJournal of Neurochemistry, 1989
- Nicotine and smoking: a perspective from animal studiesPsychopharmacology, 1987
- Development of physical dependence on morphine in respect to time and dosage and quantification of the precipitated withdrawal syndrome in ratsPsychopharmacology, 1973