Kupffer Cells Are Responsible for Producing Inflammatory Cytokines and Hepatocellular Dysfunction during Early Sepsis
- 1 May 1999
- journal article
- Published by Elsevier in Journal of Surgical Research
- Vol. 83 (2) , 151-157
- https://doi.org/10.1006/jsre.1999.5584
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- SYSTEMIC INFLAMMATORY RESPONSE SYNDROME (SIRS), MULTIPLE ORGAN DYSFUNCTION SYNDROME (MODS), MULTIPLE ORGAN FAILURE (MOF)Shock, 1998
- ANIMAL MODELS OF SEPSIS AND SHOCKShock, 1998
- Mechanism of Splenic Immunosuppression During SepsisThe Journal of Trauma: Injury, Infection, and Critical Care, 1997
- MODULATION OF KUPFFER CELL ACTIVITY BY GADOLINIUM CHLORIDE IN ENDOTOXEMIC RATSShock, 1996
- In vivo assessment of hepatic alterations following gadolinium chloride-induced Kupffer cell blockadeJournal of Hepatology, 1996
- Fluid Resuscitation Attenuates Early Cytokine mRNA Expression after PeritonitisThe Journal of Trauma: Injury, Infection, and Critical Care, 1996
- HEPATOCELLULAR DYSFUNCTION OCCURS EARLIER THAN THE ONSET OF HYPERDYNAMIC CIRCULATION DURING SEPSISShock, 1995
- ROLE OF KUPFFER CELLS IN INTERLEUKIN-6 RELEASE FOLLOWING TRAUMA-HEMORRHAGE AND RESUSCITATIONShock, 1994
- The Complex Pattern of Cytokines in Sepsis Association Between Prostaglandins, Cachectin, and InterleukinsAnnals of Surgery, 1991
- Hepatocellular dysfunction occurs early after hemorrhage and persists despite fluid resuscitationJournal of Surgical Research, 1990