INHALATION INJURY INCREASES THE ANASTOMOTIC BRONCHIAL BLOOD FLOW IN THE POUCH MODEL OF THE LEFT OVINE LUNG

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Abstract
Pulmonary parenchymal damage often occurs after airway injury. Bronchial venous drainage empties into the pulmonary microvasculature. We developed an in vivo model to study the bronchopulmonary portal system after smoke inhalation injury. Eight ewes were instrumented with hydraulic occluders on the left pulmonary artery (LPA), the left pulmonary vein, and the bronchoesophageal artery (BEA); a catheter in the LPA; and Swan-Ganz and femoral artery catheters. The vasculature between the occluders was defined as pouch. At stable mean arterial and right pulmonary arterial pressures, LPA occlusion reduced the left pulmonary artery pressure (LPAP) from 17 ± 1 mmHg to 8 ± 1 mmHg (p p
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