Cardiac hypertrophy and failure: lessons learned from genetically engineered mice
- 1 September 2001
- journal article
- review article
- Published by Wiley in Acta Physiologica Scandinavica
- Vol. 173 (1) , 103-111
- https://doi.org/10.1046/j.1365-201x.2001.00890.x
Abstract
No abstract availableKeywords
This publication has 75 references indexed in Scilit:
- Hybrid Transgenic Mice Reveal In Vivo Specificity of G Protein–Coupled Receptor Kinases in the HeartCirculation Research, 2000
- Cardiac hypertrophy with preserved contractile function after selective deletion of GLUT4 from the heartJournal of Clinical Investigation, 1999
- β-Adrenergic receptor blockade arrests myocyte damage and preserves cardiac function in the transgenic Gsα mouseJournal of Clinical Investigation, 1999
- Low- and high-level transgenic expression of β 2 -adrenergic receptors differentially affect cardiac hypertrophy and function in Gαq-overexpressing miceProceedings of the National Academy of Sciences, 1999
- Increased Protein Kinase C Activity and Expression of Ca 2+ -Sensitive Isoforms in the Failing Human HeartCirculation, 1999
- Targeted Overexpression of the Sarcoplasmic Reticulum Ca 2+ -ATPase Increases Cardiac Contractility in Transgenic Mouse HeartsCirculation Research, 1998
- Expression of protein kinase C beta in the heart causes hypertrophy in adult mice and sudden death in neonates.Journal of Clinical Investigation, 1997
- Cardiac compartment-specific overexpression of a modified retinoic acid receptor produces dilated cardiomyopathy and congestive heart failure in transgenic mice.Journal of Clinical Investigation, 1997
- Transgenic Gαq overexpression induces cardiac contractile failure in miceProceedings of the National Academy of Sciences, 1997
- MLP-Deficient Mice Exhibit a Disruption of Cardiac Cytoarchitectural Organization, Dilated Cardiomyopathy, and Heart FailureCell, 1997