And all of a sudden it's over: mitochondrial outer-membrane permeabilization in apoptosis
- 1 February 2002
- Vol. 84 (2-3) , 113-121
- https://doi.org/10.1016/s0300-9084(02)01379-2
Abstract
No abstract availableKeywords
This publication has 75 references indexed in Scilit:
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and DeathScience, 2001
- Preservation of Mitochondrial Structure and Function after Bid- or Bax-Mediated Cytochrome c ReleaseThe Journal of cell biology, 2000
- The Destabilization of Lipid Membranes Induced by the C-terminal Fragment of Caspase 8-cleaved Bid Is Inhibited by the N-terminal FragmentJournal of Biological Chemistry, 2000
- Isolation and Analysis of Components of CD95 (APO-1/Fas) Death-Inducing Signaling ComplexMethods, 1999
- Lonidamine triggers apoptosis via a direct, Bcl-2-inhibited effect on the mitochondrial permeability transition poreOncogene, 1999
- Apoptosis induced by microinjection of cytochrome c is caspase-dependent and is inhibited by Bcl-2Cell Death & Differentiation, 1998
- Anti-apoptotic oncogenes prevent caspase-dependent and independent commitment for cell deathCell Death & Differentiation, 1998
- Inhibition of Bax Channel-Forming Activity by Bcl-2Science, 1997
- Cleavage of caspase family members by granzyme B: a comparative study in vitroEuropean Journal of Immunology, 1997
- Bcl-xL forms an ion channel in synthetic lipid membranesNature, 1997