Murine DNA repair gene located on chromosome 4

Abstract
Somatic cell hybrids between primary mouse embryo fibroblast cells and SV40-transformed human XP (xeroderma pigmentosum) group A cells can express wild-type levels of DNA repair function. These hybrid cells segregate murine chromosomes in culture. The proportion of cells in a given hybrid cell line which can perform unscheduled DNA synthesis (UDS) correlated well with the percentage of the population retaining murine chromosome 4. This is the 1st example of a direct quantitative comparison of specific gene activity and chromosomal content on a cellular basis. [Xeroderma pigmentosum (XP) is an autosomal recessive human disease in which affected individuals are prone to develop skin cancers after exposure to sunlight.].