A cell cycle-dependent mechanism of renal tubule epithelial cell hypertrophy
- 1 October 1999
- journal article
- review article
- Published by Elsevier in Kidney International
- Vol. 56 (4) , 1193-1198
- https://doi.org/10.1046/j.1523-1755.1999.00708.x
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- What Makes Cells Grow Larger and How Do They Do It? Renal Hypertrophy RevisitedNephron Experimental Nephrology, 1999
- Repression of the CDK activator Cdc25A and cell-cycle arrest by cytokine TGF-β in cells lacking the CDK inhibitor p15Nature, 1997
- Cdc25 cell-cycle phosphatase as a target of c-mycNature, 1996
- Alpha Interferon Suppresses the Cyclin D3 and cdc25A Genes, Leading to a Reversible G0-Like ArrestMolecular and Cellular Biology, 1996
- NH4Cl-induced hypertrophy is mediated by weak base effects and is independent of cell cycle processesAmerican Journal of Physiology-Cell Physiology, 1996
- Involvement of pRB family in TGF beta-dependent epithelial cell hypertrophy.The Journal of cell biology, 1995
- Activation of the phosphatase activity of human cdc25A by a cdk2-cyclin E dependent phosphorylation at the G1/S transition.The EMBO Journal, 1994
- Angiotensin II-induced hypertrophy of cultured murine proximal tubular cells is mediated by endogenous transforming growth factor-beta.Journal of Clinical Investigation, 1993
- BSC-1 growth inhibitor transforms a mitogenic stimulus into a hypertrophic stimulus for renal proximal tubular cells: relationship to Na+/H+ antiport activity.Proceedings of the National Academy of Sciences, 1985
- Stimulation of Na+/H+ antiport is an early event in hypertrophy of renal proximal tubular cells.Proceedings of the National Academy of Sciences, 1985