Cellular stress inhibits vascular smooth muscle relaxation
- 1 February 2000
- journal article
- Published by Elsevier in Journal of Vascular Surgery
- Vol. 31 (2) , 343-353
- https://doi.org/10.1016/s0741-5214(00)90164-2
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Cyclosporine-induced renal artery smooth muscle contraction is associated with increases in the phosphorylation of specific contractile regulatory proteinsBiochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1999
- Thrombin activates MAPKAP2 kinase in vascular smooth muscleJournal of Vascular Surgery, 1998
- Mitogen-activated protein kinase activation: An alternate signaling pathway for sustained vascular smooth muscle contractionJournal of Vascular Surgery, 1997
- Cellular mechanisms of cyclic nucleotide-induced vasorelaxationJournal of Vascular Surgery, 1997
- Protein kinase C mediation of Ca2+-independent contractions of vascular smooth muscleBiochemistry and Cell Biology, 1996
- A novel kinase cascade triggered by stress and heat shock that stimulates MAPKAP kinase-2 and phosphorylation of the small heat shock proteinsPublished by Elsevier ,1994
- Identification of MAPKAP kinase 2 as a major enzyme responsible for the phosphorylation of the small mammalian heat shock proteinsFEBS Letters, 1992
- HSP27 is a mediator of sustained smooth muscle contraction in response to bombesinBiochemical and Biophysical Research Communications, 1991
- Direct regulation of smooth muscle contractile elements by second messengersBiochemical and Biophysical Research Communications, 1989
- Ca2+ Crossbridge Phosphorylation, and ContractionAnnual Review of Physiology, 1989