Genetic control of experimental autoimmune myasthenia gravis in mice. II. Lymphocyte proliferative response to acetylcholine receptor is dependent on Lyt-1+23- cells.
Open Access
- 1 April 1981
- journal article
- research article
- Published by Oxford University Press (OUP) in The Journal of Immunology
- Vol. 126 (4) , 1646-1647
- https://doi.org/10.4049/jimmunol.126.4.1646
Abstract
The proliferative response of regional lymph node lymphocytes to Torpedo acetylcholine receptor (TAR) is under H-2 linked Ir gene control. In high responder B6 (H-2b) congenic mice, lymphocytes that proliferate in vitro in response to TAR are dependent on T cells of a subpopulation expressing the phenotype associated with helper functions, Lyt-1+23-.This publication has 4 references indexed in Scilit:
- GENETIC-CONTROL OF EXPERIMENTAL AUTO-IMMUNE MYASTHENIA-GRAVIS IN MICE .1. LYMPHOCYTE PROLIFERATIVE RESPONSE TO ACETYLCHOLINE-RECEPTOR IS UNDER H-2-LINKED IR GENE-CONTROL1979
- HLA antigens and myasthenia gravis in JapanJournal of the Neurological Sciences, 1977
- Antibody to acetylcholine receptor in myasthenia gravisNeurology, 1976
- Detection of Anti-Acetylcholine Receptor Factors in Serum and Thymus from Patients with Myasthenia GravisNew England Journal of Medicine, 1976