A Novel Function of VEGF Receptor-2 (KDR): Rapid Release of Nitric Oxide in Response to VEGF-A Stimulation in Endothelial Cells
- 1 November 1999
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 265 (3) , 636-639
- https://doi.org/10.1006/bbrc.1999.1729
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Vascular Endothelial Growth Factor–Induced Endothelial Cell Migration and Proliferation Depend on a Nitric Oxide–Mediated Decrease in Protein Kinase Cδ ActivityCirculation Research, 1999
- VEGF-A Induces Expression of eNOS and iNOS in Endothelial Cells via VEGF Receptor-2 (KDR)Biochemical and Biophysical Research Communications, 1998
- Nitric oxide synthase modulates angiogenesis in response to tissue ischemia.Journal of Clinical Investigation, 1998
- Neuropilin-1 Is Expressed by Endothelial and Tumor Cells as an Isoform-Specific Receptor for Vascular Endothelial Growth FactorCell, 1998
- VEGF upregulates ecNOS message, protein, and NO production in human endothelial cellsAmerican Journal of Physiology-Heart and Circulatory Physiology, 1998
- Nitric oxide synthase lies downstream from vascular endothelial growth factor-induced but not basic fibroblast growth factor-induced angiogenesis.Journal of Clinical Investigation, 1997
- The Biology of Vascular Endothelial Growth FactorEndocrine Reviews, 1997
- The Vascular Endothelial Growth Factor Receptor Flt-1 Mediates Biological ActivitiesJournal of Biological Chemistry, 1996
- Endothelial Dysfunction in AtherosclerosisJournal of Vascular Research, 1996
- Placenta growth factor. Potentiation of vascular endothelial growth factor bioactivity, in vitro and in vivo, and high affinity binding to Flt-1 but not to Flk-1/KDR.Journal of Biological Chemistry, 1994