Plasticity of AMPA and NMDA receptor-mediated epileptiform activity in a chronic model of temporal lobe epilepsy

This publication has 60 references indexed in Scilit:

• Simultaneous Expression of Long‐term Depression of NMDA and Long‐term Potentiation of AMPA Receptor‐mediated Synaptic Responses in the CA1 Area of the Kainic Acid‐lesioned Hippocampus
  European Journal of Neuroscience, 1995
• Decreased heterosynaptic and homosynaptic paired pulse inhibition in the rat hippocampus as a chronic sequela to limbic status epilepticus
  Brain Research, 1993
• The Relative Contribution of NMDA Receptor Channels in the Expression of Long‐term Potentiation in the Hippocampal CA1 Region
  European Journal of Neuroscience, 1992
• Synaptic reorganization by mossy fibers in human epileptic fascia dentata
  Neuroscience, 1991
• Long-term potentiation of NMDA receptor-mediated synaptic transmission in the hippocampus
  Nature, 1991
• Glutamate decarboxylase-immunoreactive neurons are preserved in human epileptic hippocampus
  Journal of Neuroscience, 1989
• The expression of N‐methyl‐d‐aspartate‐receptor‐mediated component during epileptiform synaptic activity in the hippocampus
  British Journal of Pharmacology, 1987
• Bursting in human epileptogenic neocortex is depressed by an antagonist
  Neuroscience Letters, 1987
• Loss of inhibition in the CA1 region of the kainic acid lesioned hippocampus is not associated with changes in postsynaptic responses to GABA
  Brain Research, 1986
• Intracellular electrophysiology of CA1 pyramidal neurones in slices of the kainic acid lesioned hippocampus of the rat
  Experimental Brain Research, 1986